CRIP1 functions as an oncogene in acute myeloid leukemia by modulating glycolytic metabolic reprogramming
Muhammad Asif Zeb, Aamir Ali Khan, Ajaz Ahmad, Wentao Gui, Faryal Mehwish Awan, Bhavuk Dhamija, Sadiq Noor Khan · 2026
Read CRIP1 functions as an oncogene in acute myeloid leukemia by modulating glycolytic metabolic reprogramming by Muhammad Asif Zeb, Aamir Ali Khan, Ajaz Ahmad, Wentao Gui, Faryal Mehwish Awan, Bhavuk Dhamija, Sadiq Noor Khan (2026) in the Tower of Babel, a free online library of public-domain works preserved in clean, searchable text. No sign-up. The full text is served as a plain-text file at a predictable URL, where text is present: https://antarctic-labs.com/tower-of-babel/pmc-13243662.txt.
Description
Acute Myeloid leukemia (AML) is a stem cell-derived hematological malignancy associated with rapid progression and poor clinical outcome despite available treatments. CRIP1 exhibits tissue-specific activity, functioning as an oncogene in certain cancers while acting as a tumor suppressor in others. However, the role of CRIP1 and its association with metabolism in AML remains poorly investigated.
Catalog record
| CREATOR | Muhammad Asif Zeb, Aamir Ali Khan, Ajaz Ahmad, Wentao Gui, Faryal Mehwish Awan, Bhavuk Dhamija, Sadiq Noor Khan |
|---|---|
| YEAR | 2026 |
| COLLECTION | PAPERS |
| FORMAT | TXT |
| FILE SIZE | 37 KB |
| VERSION | 1.0 |
| LICENSE | CC BY 4.0 |
| RIGHTS | CC BY |
| SOURCE | Europe PMC |