CX3CL1 deficiency ameliorates acute kidney injury by inhibiting macrophage mitochondrial dysfunction and mtDNA-cGAS-STING signaling
Qiming Gong, Fahui Liu, Yuqing Huang, Dehui Li, Tingting Zhou, Chen Zeng, Yan Jiang, Huang Wei, Yong Xu · 2025
Read CX3CL1 deficiency ameliorates acute kidney injury by inhibiting macrophage mitochondrial dysfunction and mtDNA-cGAS-STING signaling by Qiming Gong, Fahui Liu, Yuqing Huang, Dehui Li, Tingting Zhou, Chen Zeng, Yan Jiang, Huang Wei, Yong Xu (2025) in the Tower of Babel, a free online library of public-domain works preserved in clean, searchable text. No sign-up. The full text is served as a plain-text file at a predictable URL, where text is present: https://antarctic-labs.com/tower-of-babel/pmc-12847948.txt.
Description
Qiming Gong and eight co-authors show lack of a call part, CX3CL1, eases sharp kidney harm by way of guard cell power plants. Their study appeared in twenty twenty-five. The part had tied to flare, but its role in this harm was not clear. In a germ-part mouse model, lack of the part eased kidney fail and flare.
Gene reads showed guard cell packs and power paths shift with the lack. Guard cells bent from flare-lean to calm-lean. Power plants held form, leaked less of their code, and a flare call path fed by loose code was hushed. A code guard part firms the tale.
Catalog record
| CREATOR | Qiming Gong, Fahui Liu, Yuqing Huang, Dehui Li, Tingting Zhou, Chen Zeng, Yan Jiang, Huang Wei, Yong Xu |
|---|---|
| YEAR | 2025 |
| COLLECTION | PAPERS |
| FORMAT | TXT |
| FILE SIZE | 62 KB |
| VERSION | 1.0 |
| LICENSE | CC BY 4.0 |
| RIGHTS | CC BY |
| SOURCE | Europe PMC |